To read this article in full you will need to make a payment
Purchase one-time access:Academic & Personal: 24 hour online accessCorporate R&D Professionals: 24 hour online access
One-time access price info
- For academic or personal research use, select 'Academic and Personal'
- For corporate R&D use, select 'Corporate R&D Professionals'
Subscribers receive full online access to your subscription and archive of back issues up to and including 2002.
Content published before 2002 is available via pay-per-view purchase only.
Subscribe:Subscribe to Dermatologic Clinics
Already a print subscriber? Claim online access
Already an online subscriber? Sign in
Register: Create an account
Institutional Access: Sign in to ScienceDirect
- Updates on the pathophysiology and management of acne rosacea.Postgrad Med. 2009; 121: 178-186
- Rosacea part I. Introduction, categorization, histology, pathogenesis, and risk factors.J Am Acad Dermatol. 2015; 72: 749-758
- New insights into rosacea pathophysiology: a review of recent findings.J Am Acad Dermatol. 2013; 69: S15-S26
- Integrative concepts of rosacea pathophysiology, clinical presentation and new therapeutics.Exp Dermatol. 2017; 26: 659-667
- Rosacea: the blessing of the Celts – An approach to pathogenesis through translational research.Acta Derm Venereol. 2016; 96: 147-156
- Vitamin D, vitamin D receptor, and cathelicidin in the treatment of tuberculosis.Vitam Horm. 2011; 86: 307-325
- Innate immunity and the role of the antimicrobial peptide cathelicidin in inflammatory skin disease.Drug Discov Today Dis Mech. 2013; 10: e79-e82
- Epidemiology of rosacea: updated data.Ann Dermatol Venereol. 2011; 138: S179-S183
- Risk factors associated with rosacea.J Eur Acad Dermatol Venereol. 2010; 24: 565-571
- Clinical, cellular, and molecular aspects in the pathophysiology of rosacea.J Investig Dermatol Symp Proc. 2011; 15: 2-11
- Clinical, histologic, and molecular analysis of differences between erythematotelangiectatic rosacea and telangiectatic photoaging.JAMA Dermatol. 2015; 151: 825
- Assessment of the genetic basis of rosacea by genome-wide association study.J Invest Dermatol. 2015; 135https://doi.org/10.1038/jid.2015.53
- Risk alleles for multiple sclerosis identified by a genome-wide study.N Engl J Med. 2007; 357: 851-862
- Meta-analysis identifies 29 additional ulcerative colitis risk loci, increasing the number of confirmed associations to 47.Nat Genet. 2011; 43: 246-252
- Sebaceous gland-rich skin is characterized by TSLP expression and distinct immune surveillance which is disturbed in rosacea.J Invest Dermatol. 2017; 137https://doi.org/10.1016/j.jid.2016.12.025
- TRPV4 moves toward center-fold in rosacea pathogenesis.J Invest Dermatol. 2017; 137: 801-804
- TRPV4 mediates mast cell activation in cathelicidin-induced rosacea inflammation.J Invest Dermatol. 2017; 137: 972-975
- UVB radiation generates sunburn pain and affects skin by activating epidermal TRPV4 ion channels and triggering endothelin-1 signaling.Proc Natl Acad Sci U S A. 2013; 110: E3225-E3234
- Genetic vs environmental factors that correlate with rosacea.JAMA Dermatol. 2015; 151: 1213
- Rosacea and demodicidosis associated with gain-of-function mutation in STAT1.J Eur Acad Dermatol Venereol. 2017; https://doi.org/10.1111/jdv.14413
- Clustering of autoimmune diseases in patients with rosacea.J Am Acad Dermatol. 2016; https://doi.org/10.1016/j.jaad.2015.11.004
- Sarcoidosis is associated with a truncating splice site mutation in BTNL2.Nat Genet. 2005; 37: 357-364
- Isotretinoin for the treatment of granulomatous rosacea: case report and review of the literature.J Cutan Med Surg. 2012; 16: 438-441
- Granulomatous rosacea and Crohn’s disease in a patient homozygous for the Crohn-associated NOD2/CARD15 polymorphism R702W.Exp Dermatol. 2008; 17: 1057-1058
- NOD2 is a negative regulator of Toll-like receptor 2-mediated T helper type 1 responses.Nat Immunol. 2004; 5: 800-808
- NOD2/CARD15 disease associations other than Crohn's disease.Inflamm Bowel Dis. 2007; 13: 235-241
Published online: December 16, 2017
Disclosure Statement: The authors have nothing to disclose.
© 2017 Elsevier Inc. All rights reserved.